Elaine Guo, Yuki Ishii, James Mueller, Anjana Srivatsan, Timothy Gahman, Christopher D. Putnam, Jean Y. J. Wang, Kolodner RD. FEN1 endonuclease as a therapeutic target for human cancers with defects in homologous recombination

Bin-zhong Li, Christopher D Putnam, Kolodner RD. Mechanisms underlying genome instability mediated by formation of foldback inversions in Saccharomyces cerevisiae

Srivatsan A, Li B, Sanchez DN, Somach SB, da Silva VL, de Souza SJ, Putnam CD, Kolodner RD. Essential Saccharomy cescerevisiae genome instability suppressing genes identify potential human tumor suppressors.

Graham WJ 5th, Putnam CD, Kolodner RD. The properties of Msh2-Msh6 ATP binding mutants suggest a signal amplification mechanism in DNA mismatch repair.

Srivatsan A, Li BZ, Szakal B, Branzei D, Putnam CD, Kolodner RD. The Swr1 chromatin-remodeling complex prevents genome instability induced by replication fork progression defects

Goellner EM, Putnam CD, Graham WJ 5th, Rahal CM, Li BZ, Kolodner RD. Identification of Exo1-Msh2 interaction motifs in DNA mismatch repair and newMsh2-binding partners.

Nene RV, Putnam CD, Li BZ, Nguyen KG, Srivatsan A, CampbellCS, Desai A, Kolodner RD. Cdc73 suppresses genome instability by mediating telomere homeostasis.

Bowen N, Kolodner RD. Reconstitution of Saccharomy cescerevisiae DNA polymerase ε-dependent mismatch repair with purified proteins.

Putnam CD, Srivatsan A, Nene RV, Martinez SL, Clotfelter SP, Bell SN, Somach SB, de Souza JE, Fonseca AF, deSouza SJ, Kolodner RD. A genetic network that suppresses genome rearrangements in Saccharomy cescerevisiae and contains defects in cancers.

Smith CE, Bowen N, Graham WJ 5th, Goellner EM, Srivatsan A, Kolodner RD. Activation of Saccharomy cescerevisiae Mlh1-Pms1 endonuclease in a reconstituted mismatch repair system.

Goellner EM, Smith CE, Campbell CS, Hombauer H, Desai A, Putnam CD, Kolodner RD. PCNA and Msh2-Msh6 activate an Mlh1-Pms1 endonuclease pathway required for Exo1-independent mismatch repair.

Smith CE, Mendillo ML, Bowen N, Hombauer H, Campbell CS, Desai A, Putnam CD, Kolodner RD. Dominant mutations in S. cerevisiae PMS1 identify the Mlh1-Pms1 endonuclease active site and an Exonuclease 1-independent mismatch repair pathway.